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The cholesteryl esters formed via LCAT activity are internalized into the hydrophobic core of the pre- HDL particle

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24 The oxidative stress and the increase of IOP also causes upregulation of ubiquitin (Ub) and stimulation of the Ub-proteasome pathway: 8, 25 this possibly derives from the activation of the apoptotic program

Phase 3 data suggests cagrilintide may have lower rates of nausea and vomiting compared to GLP-1 therapies like semaglutide