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The main molecular mechanisms of GLP-1RA in obesity reduction include the increased production of insulin by the cAMP/PKA pathway in pancreatic -cells, along with an augmented translocation of GLUT-4 in the cellular membrane for improved glucose homeostasis, and energy expenditure by lipolytic cycle stimulation
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It was identified in 1983 as a product of proglucagon conversion by L cells in the lining of the intestines and is also produced by pancreatic -cells, as well as being present in islet -cells and the intestine [82]
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