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In theory, increasing L-arginine availability could enhance NO production, particularly in individuals with endothelial dysfunction where NO synthesis may be impaired
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Ferrannini G, Mellbin LG, Kirabo F, Ramasundarahettige C, Gerstein HC, Rydn L
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This phenotype was manifested in the accumulation of lactic acid, pyruvate, and carnitine, increased alanine aminotransferase and aspartate aminotransferase levels in plasma [72], suppressed ETC activity, and reduced mitochondrial membrane potential ( m ) [79]
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