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D.HimaliD.et al (2023)
[11] Basen-Engquist, K., & Chang, M
Abstract Background Oxidative injury to lens epithelial cells (LECs) is directly associated with cataracts, a major contributor to blindness globally
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Metabolism abnormalities induced by exposure to excessive UA are important factors mediating the pathological development of HUA-associated multiple organ complications ( via UA excretion agent benzbromarone therapy ( via regulating transforming growth factor 1, nicotinamide adenine dinucleotide phosphate oxidase and AMP-activated protein kinase mediated reactive oxygen species and inflammation (Madlala et al., 2016
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