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B.SonmezN.CaatayP.et al
Intriguingly, the interactions of N-terminal histidine residues of the prion protein (PrP) mainly with Cu 2+ sequestration and accumulation in diseased brain (T 2+ at neutral pH (7.4) ( 2+ is not necessarily pathogenic since it has also been found to produce neuroprotection due to the cis interaction with the C terminal of PrP
Fishel, F
For research workflows that already work with each compound separately, the blend collapses three reconstitution events into one and aligns the three concentration ratios in a fixed stoichiometry
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