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A 2011 mechanism paper by Chang and colleagues in Journal of Applied Physiology demonstrated that BPC-157 promotes tendon fibroblast outgrowth, supports fibroblast survival under oxidative stress, and facilitates cell migration through the FAK-paxillin signaling pathway a pathway central to cell adhesion and movement

Additionally, its fat loss benefits could indirectly alleviate joint stress by reducing excess weight

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The lysates were collected by scraping and then subjected to sonication for 1 min