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BPC-157 is not FDA-approved for human therapeutic use

Molecular mechanisms of mitochondria-organelle interactions and ovarian physiological functions To establish a mechanistic framework for interpreting age-associated ovarian decline, this section summarizes the physiological roles of mitochondria-centered organelle interactions in oocytes and granulosa cells

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Here, working primarily in mouse hepatocytes, we show that decreasing the availability of substrate for the TCA cycle diminished NADPH production and attenuated ER stress in a manner that depended on glutathione oxidation