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Translation of the findings from MASLD/MASH animal models to humans Animal models are crucial for better understanding human MASLD pathogenesis, identifying possible therapeutic targets and disease biomarkers, as well as testing novel drugs

The researchers discovered that glutamine metabolism is impaired in the obese state, leading to increased chromatin O-GlcNAcylation and activation of genes in proinflammatory pathways

Counterargument: the higher CV is clinically insignificant for glutathione because the therapeutic window is wide

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