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These findings suggest that AST protects dopaminergic neurons through suppression of ER stress and stabilization of the miR-7/SNCA regulatory loop, presenting a promising target for PD therapy ( In summary, AST demonstrates promising therapeutic potential in PD by targeting key pathological mechanisms such as oxidative stress, neuroinflammation, and mitochondrial dysfunction

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By enhancing angiogenesis: Nutrients and oxygen reach damaged tissues faster Inflammatory byproducts are cleared more efficiently Regeneration speed increases across soft tissue, joints, and muscle BPC-157 promotes angiogenesis and capillary density in damaged tissues through modulation of VEGF and endothelial nitric oxide synthase (eNOS). Sikiric et al., Regenerative Pharmacology 2