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Mechanistically, the pathological effects of BCAAs are not attributable to a single molecular switch but rather emerge from a broader network that includes mTORC1 as one major hub, alongside impaired catabolic flux, toxic accumulation of BCKAs and branched-chain acylcarnitines, mitochondrial redox imbalance, and cellular stress pathway activation
discussion 143S-146S
Depending on your individual goals, you may either stay at 2.4 mg or decrease back to 1.7 mg as your maintenance dose
To minimize side effects and maximize benefits, both medications require a gradual dose escalation
