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Front Pharmacol 14(September):123 Kreymann B, Ghatei MA, Williams G, Bloom SR (1987) Glucagon-like peptide-1 736: a physiological incretin in man
Reductions in levels of C-reactive protein and improvements in autoimmune models suggest that semaglutide could potentially slow the progression of autoimmune diseases, such as rheumatoid arthritis and multiple sclerosis
Mark Pimentel, MD, Executive Director of the Medically Associated Science and Technology Program at Cedars-Sinai, and a leading expert on microbial overgrowths, puts it this way: Any factor that slows down the speed of flow [of food] through the small intestine has the potential to cause [bacterial] overgrowth. This is important for patients assessing their SIBO risk, because studies suggest GLP-1 signaling can inhibit aspects of the Migrating Motor Complex (MMC)
344,345 Moreover, prodrug conjugates with microbially cleavable linkers may enable site-specific drug release in tumor regions devoid of resistance-conferring microbes
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