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However, increasing oxidative stress by treating oligodendrocytes with beta-amyloid is associated with an increase in the synthesis of DHEA (284), which provides some neuroprotection in a rodent model of AD (285)

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competitive binding at the p53 TAD2 domain disrupts the FOXO4p53 interaction

Senescence Markers: Some studies explore effects on gene sets linked to cellular aging markers and proteostasis