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Modifying the delicate balance of ROS in cancer cells is an attractive method for triggering selective cell death through various pathways, such as ferroptosis, apoptosis, necroptosis, pyroptosis, parthanatos, oxeiptosis, and paraptosis

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BPC-157's documented ability to modulate the nitric oxide pathway and reduce inflammatory cytokine expression in animal models suggests a potential role in the anti-inflammatory toolkit available to longevity-focused medicine, though this remains an extrapolation requiring human validation