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PDE10A and PDE1B are highly enriched in MSNs, where they regulate D 1 /D 2 receptor-dependent cAMP/cGMP signaling and, therefore, represent potential therapeutic targets
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Together, these findings position NRF2 as the central mediator of melatonin-induced metabolic reprogramming and functional suppression in ILC2s and identify NRF2 as a therapeutically actionable target
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