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CCT-deficient B cells stimulated with LPS triggered early activation of the unfolded protein response (UPR)-mediated splicing of Xbp-1, and impaired class switching, reducing antigen-specific IgG1 production while increasing IgM secretion upon antigen challenge ( Figure 3 )

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In rat fistulas studies, creation of the fistulas by anastomoses between the different tissues, accurately provides the defined defect in each of the tissues

Both factors contribute to GSH consumption in tumor cells during anticancer chemotherapy, leading to drug resistance