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DNA damage can induce apoptosis in proliferating lymphoid cells via p53-independent mechanisms inhibitable by Bcl-2

GHK-Cu also encourages the production of other key components of the skin matrix, including elastin, proteoglycans, and glycosaminoglycans like hyaluronic acid

By highlighting the various pathways involved, including NLRP3 inflammasome activation, NF-B signaling, and autophagy/mitophagy dysfunction, this review aims to inspire further investigation into these intricate molecular processes in the absence of active viral replication in the brain

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