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Given that NACA was able to counteract the effects of the ACOX1 gain-of-function mutation, antioxidant treatment was considered for patient#1 during a severe disease flare

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These findings underscore DSIPs potential as a therapeutic agent for conditions involving mitochondrial dysfunction and oxidative stress, such as cerebral ischemia [1]

( 1 , 3 ) It works by protecting every cell in our body from free radicals and detoxifies the liver by flushing out harmful toxins