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Angiotensin-converting enzyme inhibition and angiotensin AT(1)-receptor antagonism equally improve doxorubicin-induced cardiotoxicity and nephrotoxicity
A reason for this behaviour could be that H 2 O 2 does not oxidize GSH directly, but rather through the action of glutathione peroxidase 26
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B12 plays a role in how your body processes fats and proteins, so a deficiency can leave you feeling sluggish
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