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Specifically, experimental data suggest it interacts with VEGFR2 via Src-caveolin-1-eNOS and PI3K-Akt-eNOS axes

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This age-related decline is one of the most frequently cited data points in copper-peptide research literature, and its a primary reason GHK-Cu is studied within the broader context of cellular aging and peptides for longevity research rather than treated purely as a wound-healing or dermatology compound