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The equivalent of a thallium binding residue from an archeal homolog controls cation interactions in brain glutamate transporters

These drugs mimic the action of naturally occurring GLP-1, a hormone produced in the intestines in response to food intake

The ULK1 complex acts as a key signaling hub, mediating stress-induced AMPK signaling transduction, regulating the mitochondrial autophagy process through phosphorylation [44]

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