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Consistently, ACC inhibition (ND630) also modestly increased histone acetylation and proliferation in the DKO cells while suppressing malonyl-CoA levels (Supplemental Figure 4D, E, F), indicating that fatty acids may act to increase histone acetylation in part through acetyl-CoA sparing from ACC consumption

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3.3 Network of inflammatory mediators The network of inflammatory mediators after ischemic stroke is a complex regulatory system composed of various molecules, including cytokines, chemokines, the complement system, and metabolites (97)

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