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Conclusion: Given the limited epidemiological knowledge on MMNSTs, our study contributes to the literature by documenting a case of intra- and extraspinal, cervical MMNST without any of the previously known driver mutations or copy number changes
Additional evidence also indicates that activation of the GLP-1/GLP-1R signaling pathway is implicated in the pathogenesis of sepsis-associated encephalopathy (SAE)
The same antibody is also being tried with semaglutide in a worldwide Phase II clinical trial run by Eli Lilly
Glucagon-like peptide-1 suppresses neuroin flammation and improves neural structure
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