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Evidence from animal models points toward support of axonal continuity, synaptic stability, and functional recovery following nervous system injury

The interactions between citrulline and molecules associated with the JAK/STAT axis, together with the regulatory effects of urea cycle-related enzymes on the activation of inflammatory macrophages, suggest that the iNOS/NO axis may exert feedback regulation on pro-inflammatory signaling in different contexts ( At the intervention level, regulating the metabolic partitioning of Arg can alter macrophage phenotypes and inflammatory intensity

Pregnant individuals, people with chronic medical conditions, and patients taking prescription medications should consult a healthcare provider to evaluate potential risks or interactions

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