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Concurrently, damaged mitochondrial DNA fragments are released into the cytoplasm, activating the stimulator of interferon genes signaling pathway, which further induces apoptosis of pancreatic -cells
It remains to be determined whether the accumulation of Nrf2 and excessive reductive stress also underlie the impaired MuSC functions after a round of self-renewal
BPC-157 is generally well-tolerated, with minimal side effects
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