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Cassani B, Villablanca EJ, De Calisto J, Wang S, Mora JR
BPC-157s primary signalling mechanisms are well-characterised: it activates VEGFR2, drives downstream ERK1/2 phosphorylation, and stimulates nitric oxide synthesis via the Akt-eNOS axis producing pro-angiogenic, fibroblast-activating, and extracellular matrix-stabilising effects in experimental models
The fact that these substances are banned is certainly not proof that they work
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