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It remains uncertain whether distinct initiation mechanisms trigger the inflammatory response by acting on priming signaling pathways, or if a reduction in the negative regulation of NLRP3 activation amplifies the inflammatory cascade
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(2019) Cell Tissue Res Sikiric et al
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From that biology, researchers draw a theoretical concern: chronically potentiating a proliferative, pro-angiogenic pathway is a plausible mechanism by which an agent could, in principle, support growth of existing malignancy
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