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Neuronal circuitry mechanism regulating adult quiescent neural stem-cell fate decision
AOD9604 did not stimulate the production of IGF-1 in any of the systems studied, and elicits its fat-reducing effects without inducing insulin resistance a key pharmacological distinction from intact hGH that makes it a uniquely clean research tool for studying lipolytic mechanisms in isolation
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316 In KRAS/LKB1-mutant NSCLC, resistance to KRAS inhibitors such as adagrasib involves adeno-to-squamous transdifferentiation (AST), driven by Np63 and characterized by KRT6A expression
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