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Clinical evidence indicates that aberrant airway colonization by specific bacteria, including Moraxella and Lactobacillus , relieves the endogenous post-transcriptional inhibition of IL-17A by downregulating non-coding RNAs such as miR-122 and miR-30a ( Foxp3 promoter region, antagonizing RORt transcriptional activity and inhibiting IL-17A expression at the chromatin level ( 2.1.3 Downstream signal transduction and post-receptor escape mechanisms IL-17A signals through high-affinity binding to the heterodimeric receptor IL-17RA/RC, recruiting the adaptor protein ACT1 via the intracellular SEFIR domain (64)
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