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Three mechanistic explanations, not mutually exclusive: Signalling cascade persistence: BPC-157 activates VEGFR2 and FAK-paxillin pathways whose downstream effects (angiogenesis, fibroblast migration, collagen remodelling) unfold over days to weeks after initial receptor engagement
Adequate studies in humans have not been conducted
Organized fiber architecture directly correlates with higher tensile strength and reduced re-injury risk at the repair site
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