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Therapy should limit pathological lactate accumulation during acute ischemia while preserving astrocyte-neuron and endothelial-pericyte lactate shuttling during repair [11,12,13,14]
Their activation, therefore, impacts brain physiology at many levels including neural circuits, cell populations, synapses, and neurotransmitter signaling (Bohlen et al., 2019
A powerful, natural antioxidant, glutathione has been the object of thousands of studies, many of which have investigated its protective effects and therapeutic potential
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