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doi: 10.1016/j.bone.2013.06.029 55 YamagishiS

Life Sci

10.1016/s0742-8413(96)00223-x 99 SmithE.OttossonF.HellstrandS.EricsonU.Orho-MelanderM.FernandezC.et al (2020)

Gallen Formel-E Zrich Diplomfeier WBZ ETH Fest Fielmann, St

It may cause damage and apoptosis of TECs through multiple mechanisms, including mitochondrial dysfunction, oxidative stress, regulation of HIF-1, and alteration of extracellular matrix ( 3.4 Inflammation Inflammation plays a crucial role in the pathophysiology of DKD