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However, its antioxidant properties may help reduce oxidative damage to melanocytes, potentially slowing the process

Damaged AECs release pro-inflammatory and pro-fibrotic mediators, including interleukin (IL) -1 and tumor necrosis factor (TNF) -, which promote fibroblast recruitment, inflammatory cell activation (e.g., neutrophils, Th17, gdT cells), and myofibroblast differentiation, ultimately leading to ECM accumulation and fibrotic tissue remodeling [20, 29, 33, 34]

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2009 Jan;37(1):47-51

In one form of hormesis, sublethal exposure to stressors induces a response that results in stress resistance