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Doses in preclinical studies have typically ranged from 10 nanograms to 10 micrograms per kilogram of body weight, administered daily

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In the acute phase, METH causes an excessive release of dopamine and glutamate, resulting in transient deficits in attention and working memory (Mark et al., 2004)

Dihexa, with its combined N-hexanoic-Tyr-Ile-(6) aminohexanoic amide structure, exhibited a significantly extended half-life of 335.5 9.5 min, confirming that both N- and C-terminal modifications are effective strategies for improving metabolic stability [2]

[ | ] [ | ] Charnay Y, Bouras C, Vallet PG, Golaz J, Guntern R, Constantinidis J (1989)