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There are several known mechanisms that explain its neuro-protective effects: It plays a pivotal role in combating oxidative stress, reducing inflammation, and promoting mitochondrial function, and it is a precursor to glutathione
Only about 1% of oral B12 is absorbed passively when intrinsic factor is absent or impaired
M.Morales VillegasE
The timeline: when does it start, when does it stop
Interestingly, hyperplasia of selected EEC subtypes in response to intestinal infection is probably dependent on immune cells, since serotonin (5-HT)-producing cell hyperplasia associated with Citrobacter rodentium infection was absent in mice lacking adaptive immunity ( + T cells were necessary to increase numbers of 5-HT producing enterochromaffin cells during helminth infection (149)
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