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Research data suggest that this combination significantly accelerates protein synthesis, stimulates both hypertrophy and hyperplasia of muscle cells, supports bone density, and dramatically shortens the time required for the repair of damaged tendons, ligaments, and joint structures

It acts at GHS-R1a, whose endogenous ligand the acylated 28-residue gastric peptide ghrelin was identified in 1999, a year after ipamorelin was first described [2]

Studies published in Wound Repair and Regeneration have shown that GHK-Cu treatment of keloid fibroblasts increases MMP-2 activity by 30-50%, promoting degradation of excessive collagen deposits

Plates were washed with PBS-T and anti-mouse IgM-HRP or anti-mouse IgG-HRP (Sigma A8786 and A3673) was used for detection