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Stem Cell Res Ther
doi:10.1172/JCI116186 Willms B, Werner J, Holst JJ, Orskov C, Creutzfeldt W, Nauck MA
Contrastingly, the expression of SMR1 was robustly reduced at both concentrations (Fig
Tendon injuries initially suffer from: Poor blood supply Limited vascularity Slow nutrient delivery In rodent models, BPC-157 appears to: Upregulate VEGFR2 signalling Increase capillary density Improve microvascular perfusion Mechanistically: BPC-157 VEGFR2 activation PI3K/Akt pathway eNOS phosphorylation Increased nitric oxide Improved angiogenesis More vascularity = better nutrient and oxygen delivery to fibroblasts
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