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reported that the expression levels of ALDH2 decreased in mice after long-term alcohol intake, leading to the accumulation of toxic aldehydes and accelerating liver inflammation and fibrosis (Gao et al., 2019b)

Standard treatmentsrest, ice, anti-inflammatories, and physical therapyprovide symptom relief but do little to address the underlying hypovascularity

They may include increased GSH utilization upon exposure to oxygen, insufficient GSH synthesis due to immature synthesis enzymes or to reduced availability of cysteine, the rate-limiting precursor amino acid for GSH synthesis, or impaired regeneration of oxidized glutathione (GSSG) to GSH

To accomplish this, researchers used 32 male New Zealand white rabbits, each 12 weeks old