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Excess adipose tissue promotes inflammation, alters hormone levels, and creates a metabolic environment associated with increased breast cancer risk, particularly after menopause
An important theoretical concern exists: c-Met is overexpressed in multiple human cancers (gastric, lung, colorectal), and chronic c-Met pathway stimulation could potentially influence cancer riskthough this has not been studied longitudinally even in animals
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Nevertheless, the interpretation of their therapeutic potential remains preliminary
