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McCurley, A

glucose tolerance were maintained in Gipr AP-KO mice as expected, consistent with direct effects of acyl-GIP on pancreatic -cells rather than an indirect effect mediated via the AP (Extended Data Fig

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Diabetes 120 , 132138 (2012)

Key findings include: Upregulation of VEGFR2 expression and internalization in endothelial cells Activation of the VEGFR2-Akt-eNOS signaling cascade Enhanced endothelial cell proliferation and tube formation Improved collateral circulation in ischemic injury models The peptides pro-angiogenic effects appear particularly relevant in healing scenarios where blood supply restoration is critical for tissue recovery