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The mechanisms underlying IGF-1 LR3's anabolic effects involve activation of the PI3K/Akt/mTOR signaling pathway, which stimulates ribosomal protein S6 and translation initiation factors downstream of mTORC1, thereby enhancing protein synthesis
Recent studies have shown that short-term IGF-1 treatment (1 g/g/day) using an osmotic minipump for 3 days after acute myocardial infarction improves cardiac function by modulating the acute inflammatory phase but does not alleviate fibrosis [27]
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Additionally, GLP-1 receptor activation suppresses glucagon secretion from pancreatic alpha cells, reducing hepatic glucose production
doi: 10.3389/fnut.2022.811469 126 RusnakTAzarcoya-BarreraJWollinBMakarowskiANelsonRFieldCJet al
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