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Water follows the electrolytes, leading to cell shrinkage, a hallmark of eryptosis [90]
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Key physiological features include: Filaggrin gene mutations , leading to a compromised skin barrier and increased transepidermal water loss Th2-skewed immune response , with elevated IL-4, IL-13, and IgE levels Colonization with Staphylococcus aureus , often contributing to inflammation and secondary infection Heightened mast cell activity , promoting pruritus and dermal reactivity This pathophysiology supports a dual treatment approach: barrier repair and inflammation modulation , often in combination with antimicrobial or immunosuppressive strategies
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Thus, residues Tyr110 and Lys105 might share a function in accordance with the glutathione activator model but might also differ, for example, regarding the protonation state of the active site cysteine residue
