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Importantly, wild-type BoNT/X protease did not induce notable apoptotic or lytic cell death (Extended Data Fig

Trimethylamine N-oxide aggravates human aortic valve interstitial cell inflammation by regulating the macrophages polarization through a N6-methyladenosine-mediated pathway

on BDI and MADRS, effects favored ALC but varied among studies

Studies suggest the compound may enhance the brain's intrinsic capacity for repair through mobilization of existing plasticity mechanisms rather than requiring exogenous cell replacement therapies