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Injections bypass gut absorption problems and deliver a predictable dose into muscle or subcutaneous tissue

Improved Healing Animal studies indicate that BPC-157 stimulates the proliferation of fibroblasts to enhance healing in many tissue types

Cardiomyocyte survival promotion through ILK/PI3K/Akt anti-apoptotic signalling, cardiac progenitor cell activation and migration toward the ischaemic injury border zone, angiogenesis stimulation in the peri-infarct region to restore oxygen delivery to at-risk myocardium, and anti-fibrotic effects that attenuate the non-functional scar tissue formation that impairs cardiac function following myocardial infarction

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Though much of the research around PDA and BPC-157 remains preliminary, studies and clinical observations suggest the following mechanisms: 1