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The activation of PPAR- upregulates the transcription of acyl-CoA oxidase 1 (ACOX1), and a range of substances, including PFOS, PFOA, PFNA, and PFHxS, can act as the predominant activates of PPAR-, interfering with peroxisomal -oxidation, leading to hepatocyte mitochondrial damage and lipid accumulation [76, 77]

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