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10.3892/or.2020.7582 283 LiuX.XieC.WangY.XiangJ.ChenL.YuanJ.et al (2024b)
demonstrated the effect of glucagon to stimulate beta-cell cAMP production specifically through the GCGR ( 2+ levels, and ERK1/2 activation ( 2+ levels in response to glucagon treatment, and dose-dependent activation of ERK1/2 ( Considering the functional and structural similarities between the GCGR and GLP-1R, it was predicted that glucagon mediated these insulinotropic effects through signaling at the beta-cell GCGR
It reduces angiotensin levels by targeting the synthesis of angiotensinogen (AGT), the most upstream component of the renin angiotensin aldosterone system (RAAS), a classic pathway for blood pressure regulation
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