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Unlike apoptosis, paraptosis does not hinge on caspase activity but frequently hinges on the activation of mitogen-activated protein kinases (MAPKs), such as MAPK8/JNK, MAPK14/p38, and mitogen-activated protein kinase kinase 2 (MAP2K2/MEK2) [148]

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This protective mechanism is particularly effective in preserving mitochondrial function and vascular responsiveness, two critical factors in the pathophysiology of sepsis-related organ failure (Salvemini and Cuzzocrea, 2003)