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Studies suggest that KPVs anti-inflammatory mechanism is independent of melanocortin receptors and involves direct entry into cells, distinguishing it from full-length -MSH signaling
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In our model, depletion of Hypoxia-Inducible Factor 1 alpha (HIF-1) impaired the effects of Exendin-4 on glucose metabolism, while pharmacological inhibition of Phosphoinositide 3-kinase (PI3K) or mTOR completely abolished such effects
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