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While preclinical studies report associations of interest for metabolic research, human data are absent
2012), our observation that APAP decreased GJ abundance suggested that hepatocytes mount a defense against the spread of oxidative stress molecules within liver tissue during the early stages of APAP exposure by reducing the expression of Cx32 (and consequently GJs)
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Results of experimental and clinical investigations demonstrated the effects of the synthetic peptide on the course of retinitis pigmentosa, a degenerative process of the retina resulting from disturbed metabolism of specific proteins in the pigmented epithelium and other retinal layers [38]
Further, dihexa facilitated memory consolidation and retrieval in the scopolamine-induced amnesic rat model of AD, as well as in memory compromised aged rats as measured using the Morris water maze task of spatial memory [73]
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