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Moreover, developing molecules or chimeric drugs capable of simultaneously blocking multiple cell death pathways may be promising DKD therapeutic approaches, potentially providing patients with more precise and effective treatment options

Endothelial dysfunction, including decreased endothelial cell-dependent vasorelaxation, is increasingly proposed to be triggered by free radicals generated during auto-oxidation of homocysteine (Loscalzo, 1996)

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Both of these conditions are a result of the interaction of multiple genetic factors and environment, involving multiple genes with small interactive and additive effects