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IGF-1 SIGNALING IN THE MODULATION OF ANABOLIC RESPONSES IN BONE In this section, we shall highlight two mechanisms by which IGF-1 exerts its action on the skeleton, specifically, by modulating the anabolic effects of parathyroid hormone (PTH) and through crosstalk with mechanosensing pathways
The proposed mechanism involves preventing GABA receptor downregulation, counteracting compensatory NMDA receptor sensitization, and stabilizing benzodiazepine receptor configuration
doi: 10.1002/jmv.24692 18
10.1016/s0016-5085(98)70012-5 86 ThielemansL.DepoortereI.PerretJ.RobberechtP.LiuY.ThijsT.et al (2005)
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